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12th Congress of the European Hematology Association
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Prof. R. Bertina
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Leiden, Netherlands 
University Medical Center Leiden
Rogier M. Bertina PhD is head of the Haemostasis and Thrombosis Section of the Department of Haematology at the Leiden University Medical Center, since 1995. He joined the scientific staff in 1975 and became Professor in Biochemistry of Blood Coagulation in 1989. He was trained in biochemistry at the University of Amsterdam and completed his PhD Thesis in 1972 at the same university. He is (co)author of more than 370 papers in the field of Haemostasis and Thrombosis. His main interest is in the area of hereditary coagulation defects. Initially he focussed on the haemophilias, later on the thrombophilias. His group was among the first to report on hereditary deficiencies of protein C and protein S, and for the first time used mutation analysis in the study of these deficiencies. His group first reported the mutation in the factor V gene (factor V Leiden) that is associated with APC-resistance and more recently the mutation in the prothrombin gene (G20210A) that is associated with increased risk for venous thrombosis. His present interest is the identification of novel genetic risk factors of thrombosis, the study of their interactions and their downstream effects.
Since 1985 he is scientific director of RELAC, a foundation active in the field of prothrombin time standardization. He was the first president of the Dutch Society of Thrombosis and Haemostasis (1988-1991) and member of the ICTH (1983-1989) and the Council of the ISTH (1994-2000). He received the ISTH Investigator Recognition Award in 1995, and the ISTH Distinguished Career Award in 2001.
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| Presentations by Prof. R. Bertina currently available on MultiWebcast.com |
12th Congress of the European Hematology Association
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Thrombophilia: from single gene disorder to complex disease
2007-06-08 - English - Thrombosis
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Venous thrombosis (VT) is an episodic disease. Blood clots are formed in veins which obstruct the circulation of the blood locally or, after the formation of emboli, elsewhere in the circulation. Acute events can be treated effectively by anticoagulant treatment aiming at...(more) |
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